SOCS1 and glycogen storage disease VI: In the presence of NahG, spontaneous lesion formation was suppressed in the lesion mimic mutants lesion simulating disease 6 (lsd6), lsd7, accelerated cell death 5 (acd5), acd6, acd11, constitutive expressor of PR genes 22 (cpr22), ssi1, and disease-like lesions 1 (dll1), suggesting that SA plays an important role in spot-like formation [43,44,45,46,47,48,49].