Previous studies showed that FA-AKI increases serum proinflammatory cytokines IL-1β, IL-5, IL-6, IL-18, interferon-γ, TNF-α, MCP-1, and fibrosis growth factor 23 (FGF23) [34,35,36,37] that may trigger inflammation in the heart. The gene discussed is CCL2; the disease is acute kidney injury.