Cardiovascular risk factors such as high glucose and dyslipidemia activate the vascular endothelium to secrete inflammatory chemokines (MCP1 and IL8) and adhesion molecules (ICAM1, VCAM1, and E-Selectin), which leads to monocyte binding to the vasculature and the subsequent vascular inflammation [3,13]. This evidence concerns the gene CXCL8 and metabolic syndrome.