Finally, Papagiannakis et al. [108] described increased dimeric and dimeric/monomeric ratios of aSyn in the erythrocyte membranes from patients with PD carrying glucocerebrosidase mutations and in patients with genetically undetermined PD, but not in PD patients carrying the A53T mutation in the SNCA gene, compared with HCs, while monomeric aSyn levels did not differ significantly between the three groups of PD patients and HCs. This evidence concerns the gene GBA1 and Parkinson disease.