Ren et al. [107] demonstrated that miR-210-3p is overexpressed in bone metastatic PCa tissues and promotes sustained activation of the NF-κB signaling pathway by inhibiting TNFAIP3 interacting protein 1 (TNIP1) and suppressor of cytokine signaling 1 (SOCS1) expression, negative regulators of NF-κB signaling. Here, TNIP1 is linked to posterior cortical atrophy.