The findings that HAGLR is expressed at high levels in cervical cancer cells compared to control [38,42], that knockdown of HAGLR significantly inhibited cervical cancer cell proliferation and colony formation, and that cervical cancer cell growth can be suppressed by inactivating the Ras/ERK pathway [38,42] reinforce our conclusion of the importance of HAGLR as a target for breast and cervical cancer. Here, HAGLR is linked to cervical carcinoma.