Of note, navitoclax and venetoclax also upregulated the expression of the chemokine receptors CCR7 and XCR1, as well as the maturation marker MHC-II, to a variable degree on cDC1 cells (defined as CD103+CD11b- cells within DCs, defined as the viable CD45+CD11c+F4/80- MHC-IIhi population, Fig. S6A) in the blood, tumor infiltrate, and lymph nodes from mice bearing TC1 orthotopic lung cancers (Figure S6B-D), without any signs of a reduction in absolute numbers of such DCs in those organs (Fig. S6E-G). This evidence concerns the gene PTPRC and lung cancer.