Other proteinases from the ADAMTS family have also been implicated in a myriad of cardiovascular pathologies, e.g., ADAMTS2 expression was predominantly elevated in failing human hearts [58]; the inhibition of re-endothelialization and formation of coronary plaques leading to ischemic CVD due to ADAMTS7 [59]; and elevated levels of ADAMTS7 in patients with LVEF ≤35% compared with those with LVEF >35% after acute myocardial infarction, which was independent of traditional cardiovascular risk factors and other biomarkers [60], just to enumerate a few. This evidence concerns the gene ADAMTS7 and acute myocardial infarction.