TGFB1 and pulmonary fibrosis: Multiple murine models of pulmonary fibrosis display evidence of various cellular senescence markers, including heightened aging-associated β-galactosidase in lysosomes, an increased BCL-2/Bax ratio of apoptosis-involved proteins in mitochondria, and amplified DNA damage in the nucleus (Schafer et al., 2017; Blokland et al., 2020), all coupled with robust pro-fibrotic effects, such as TGF-β (Rana et al., 2020).