IL6 and COVID-19: The overproduction of several inflammatory mediators such as interleukin 1β (IL-1β), interleukin-2R, interleukin 6 (IL-6) up to interleukin 10, interferon-γ, monocyte chemoattractant protein 1A, macrophage inflammatory protein 1A/B, tumor necrosis factor-α, and vascular endothelial growth factor; overactivated and exhausted T-cells; and a rise in autoantibodies characterize the immune system’s hyperstimulation in COVID-19 [20,23,24,25].