They also reported that only HSCs expressing the KGF factor mitigated the histological signs of bleomycin-induced lung fibrosis, stimulated the proliferation of alveolar type II cells (AEC2s), and decreased α-smooth muscle actin (α-SMA) positive myofibroblasts, chemokines, and pro-inflammatory cytokines (64). The gene discussed is ACTA1; the disease is pulmonary fibrosis.