With regard to FATP4, we demonstrated that in the adipocytes obtained from the subADMSCs, three of the examined factors—AS160 silencing (33% of data variability, p < 0.05), obesity (29% of data variability, p < 0.05), and the presence of metabolic syndrome (32% of data variability, p < 0.05)—influenced the plasmalemmal expression of the FATP4 protein (Figure 5C). This evidence concerns the gene SLC27A4 and obesity due to melanocortin 4 receptor deficiency.