VWF and von Willebrand disease (hereditary or acquired): Although not really VWD, since defects lie in the platelet GPIb receptor, inclusion of “platelet type” [PT] or pseudo-VWD in discussions related to testing is helpful, since patients present phenotypically similar to those with type 2B VWD (ie, gain-of-function GPIb leads to “spontaneous” binding to normal plasma VWF, which may lead to clearance of both (HMWM) VWF and platelets from circulation (ie, lower residual VWF activity and [mild] thrombocytopenia) [6].