Furthermore, EBLN3P can modulate the proliferation of lung adenocarcinoma cells through the miR-655-3p/Bcl2 axis.36 Knockdown of EBLN3P hinders the proliferation, invasion, and migration of Jurkat cells by increasing miR-655-3p expression.37 As for HuR, there is evidence that HuR-RNA interactions play essential roles in cancer treatment.38 Additionally, the interplay between HuR and microRNAs in gene regulation is implicated in angiogenesis.39 There is no report on the regulation of lncRNA EBLN3P on HuR. The gene discussed is ELAVL1; the disease is cancer.