This age‐related phenomenon is corroborated by Kumar et al. (2018), who showed that FDG‐PET signals in the interictal period (confirmed by simultaneous EEG during the FDG uptake period) were normal in children with Dravet syndrome with confirmed SCN1A variants aged 6 months to 1 year (n = 3) compared to age‐matched epileptic controls (n = 3), but when the children with Dravet syndrome were followed up at age 3–5 years, cortical FDG‐PET signals were found to be lower compared to a separate group of age‐matched epileptic controls (n = 3). Here, SCN1A is linked to encephalopathy, progressive, early-onset, with brain edema and/or leukoencephalopathy.