However, a role for adipokines and insulin resistance in pathogenesis of steatosis does not occur at least in the conditions evaluated in our study, since levels of glucose, insulin, and adiponectin were similar in plasma at 4 h of reperfusion in experimental groups BD+IL-6+LT, BD+IL-10+LT, BD+anti-IL-6+LT, BD+anti-IL-10+LT, and BD+LT, with steatotic and non-steatotic liver grafts from DBDs (data not shown). The gene discussed is INS; the disease is Insulin resistance.