To identify m6A readers involved in METTL3-induced SCAP translation, we performed knockdown of m6A readers (YTHDF1/2/3, YTHDC1/2, and IG2BP1/2/3) in NAFLD-HCC cells overexpressing METTL3 (Figure S5K). This evidence concerns the gene METTL3 and metabolic dysfunction-associated steatotic liver disease.