found that overexpression of miR‐411 in the heart significantly improved the heart phenotype after myocardial infarction and in this process, miR‐411 directly regulated Foxo1 expression and then enhanced YAP activity and nuclear translocation, ultimately promoting myocardial cell proliferation and inhibiting myocardial cell apoptosis. Here, YAP1 is linked to myocardial infarction.