In another study, they silenced TLR10 in the human colon adenocarcinoma cell line HT-29 and in monocytic THP-1 cells, which led to increased viability of Listeria monocytogenes, and they unveiled that the heterodimer of TLR2/TLR10 was the one behind the activation of NF-κB [57]. This evidence concerns the gene TLR10 and colon adenocarcinoma.