To further determine, on the one hand, whether the antiapoptotic effects observed with IMT504 were specific of MIN6B1 cells or more general of β‐cells and, on the other hand, to determine whether another β‐cell proapoptotic stimulus typical of T1D, such as a cytokine mix, could also be reversed by IMT504, Beta‐TC‐6 cells were incubated for 24 h with a cytokine mix (TNFα, IL‐1β, IL‐6, and INFγ) in the presence or absence of IMT504 (4 or 8 μg/mL) and Annexin V to determine the apoptotic nuclei. Here, IL6 is linked to type 1 diabetes mellitus.