DAF was unchanged in both the obese and nonobese PCOS compared to their respective controls and between the obese and nonobese PCOS cohorts, which is surprising, as it acts to accelerate the decay in the classical and alternative C3 and C5 convertases [27] and, as is shown here, there were changes in C3 and C5. This evidence concerns the gene C3 and polycystic ovary syndrome.