In a murine model of ovalbumin-induced pulmonary inflammation, CCL24 knockout mice exhibited reduced cell infiltration into the bronchoalveolar lavage fluid, indicating a significant decrease in inflammation.56 Similar results were also evident in a bleomycin-induced skin-and-lung inflammation and fibrosis mouse model, using CCL24 knockout mice.57 This suggests that CCL24 contributes to the inflammatory response seen in these models, highlighting its pro-inflammatory function in the context of pulmonary inflammation. This evidence concerns the gene CCL24 and fibrosis.