Additionally, ApoA1 and ApoB levels were lower in AAA rupture patients than in non-rupture patients (0.77 ± 0.17 vs. 0.97 ± 0.23, P < 0.001; 0.67 ± 0.20 vs. 0.80 ± 0.25, P = 0.009). This evidence concerns the gene APOB and triple-A syndrome.