LGALS3 and neoplasm: Of note, Mertk−/− or Lgals3−/− mice were found to be defective in their ability to support starry‐sky lymphoma growth243, 277 and the gene expression profiles of SS‐TAMs were predicted not only to signal effective uptake and anti‐inflammatory/immunosuppressive responses to apoptotic tumor cells but also potentially to produce trophic, angiogenic, and invasive factors for tumor growth and evolution.276