Recently, by integrating an analysis of whole-genome and transcriptome sequencing datasets of two PCa cohorts [8, 13] with a collected genome-wide CRISPR/Cas9-mediated loss-of-function screen data in PCa cells [14], we observed that GATA2 is top-ranked among all other copy number amplified potential causal genes, making it an interesting target to further explore its disease predictive and causal roles in PCa. This evidence concerns the gene GATA2 and posterior cortical atrophy.