STAT3 and neoplasm: CD47 expression can be regulated at the transcriptional level by multiple transcription factors, including signal transducer and activator of transcription 3 (STAT3),[4] β‐catenin–transcription Factor 4 (TCF4),[5] hypoxia‐inducible factor 1 (HIF‐1),[6] and c‐Myc.[7] However, whether CD47 expression is regulated at the posttranslational level, especially in response to tumor‐frequently occurred receptor tyrosine kinases (RTKs) lesions, thereby contributing to tumor immune evasion remains unclear.