We suggest a direct causal link between the identified CSF GFAP-Abs and such phenotype for the following reasons: (1) GFAP was found to be expressed in the DRG, and a patient’s CSF was able to stain the DRG by immunofluorescence [4, 9], (2) autoimmune processes are frequent in sensory neuronopathies, and several other antibodies have already been described in this syndrome such as Hu, CV2/CRMP5, FGFR3, or AGO antibodies [20–23], (3) the patient presented herein had a lung adenocarcinoma found on tumor screening, suggesting a possible immune paraneoplastic origin [23]. The gene discussed is FGFR3; the disease is neoplasm.