VIM and breast carcinoma: Since EMT has been implicated for breast cancer metastasis, angiogenesis, drug resistance and eventually cancer stem cell formation [7] [8] [9], we in this study investigated the differential gene expression and core canonical pathways involved in the EMT changes in a TNBC-EMT model Vimentin-RFP knock in cell line(ATCC HTB-26MET) in comparison to the same non EMT TNBC MDA-MB-231 breast adenocarcinoma cell line (ATCC HTB-26) by analyzing the transcriptomic data obtained by NGS RNA Sequencing and using the Ingenuity Pathway Analysis (IPA) Software as licensed by Qiagen Corporation, USA.