SMARCA4 and granulosa cell tumor: Only three cellular models are currently available to study the molecular pathogenesis of SCCOHT, including: (1) BIN-67 cell line, derived from metastatic pelvic nodes in primary SCCOHT [10]; (2)SCCOHT-1 cell line [11], derived from the SCCOHT that relapsed after chemotherapy; (3) COV434 cell line, which is originally described as a granulosa cell tumor [12–14], recently re-revised as a SCCOHT cell line due to the presence of a mutation in the SMARCA4 gene and in combination with the clinical features of patients from whom this cell line originated [15].