The results indicated that the higher abundance of PARK7+NK-C4, DYNC1H1+CD8+ T−C2, DYNLL1+Mac-C1, and TUBA1A+Mac-C3 were observed in the AML samples, while the higher abundance of TUBA1B+CD8+ T−C1, UBE2V1+CD8+ T−C3, and UBE2V1+Mac-C2 were observed in the normal samples (Figure 5A). The gene discussed is TUBA1A; the disease is acute myeloid leukemia.