We did not examine the impact of the mPGES-1 inhibitor on the infiltration of immune-suppressive cell populations—including regulatory T cells, myeloid-derived suppressor cells, and tumor-associated macrophages—because our earlier study has already shown that genetic depletion of mPGES-1 did not suppress the number of tumor-infiltrating immune-suppressive cells (25). Here, PTGES is linked to neoplasm.