Furthermore, we also analyzed the RNA‐seq data of cohort GSE167514 (human RCC cell lines 786O transfected with GDH1‐shRNA and Control‐shRNA) and GSE121580 (human glioma cell lines U87 transfected with GDH1‐shRNA and Control‐shRNA).[13] Consistently, antigen presentation and processing pathways were enriched and B2M was elevated in the ShNT group with higher αKG levels (Figure S1C‐G, Supporting Information). This evidence concerns the gene GLUD1 and glioma.