Unlike Mettl14Δfat mice which are resistant to HFD‐induced obesity, mice with BAT‐specific deletion of Mettl3 are prone to diet‐induced obesity.[18] These opposite phenotypes further support the notion that increases in β adrenergic signaling and lipolysis in WAT, which are observed in Mettl14Δfat mice but not in Mettl3‐deficient mice, mediate protection of Mettl14Δfat mice against obesity. Here, METTL3 is linked to Obesity.