In particular, obesity increases the risk of critical illness from COVID‐19 by 97%,2 which could be explained by constitutive up‐regulation of pro‐inflammatory factors, compromised leukocyte function (due to lipid deposition in lymphoid tissue) and over‐expression of Angiotensin Converting Enzyme‐2 (ACE‐2) (the transmembrane enzyme exploited by SARS‐CoV‐2 for cell penetration).3 The gene discussed is ACE2; the disease is COVID-19.