BAK1 and B-cell non-Hodgkin lymphoma: The anti-apoptotic protein Bcl-2 homologous antagonist/killer (BCL-2), overexpressed in some subtypes of B-cell lymphomas, regulates mitochondrial function and mediates therapeutic resistance by inhibiting the oligomerization of the pro-apoptotic Bcl-2 homologous antagonist/killer (Bak) and Bcl-2-associated X protein (Bax) proteins and by facilitating the import of GSH and complex IV subunits to mitochondria (Mattes et al., 2019).