It showed increased abundances of the AP‐3 complex subunit AP3B1, somatic mutations of which confer higher risk for severe COVID‐19 (Luo et al, 2021) as well as the collagen‐associated protein PLOD3, which is associated with common pulmonary fibrosis (Shao et al, 2020; exemplary shown in Fig 3D and E). This evidence concerns the gene AP3B1 and COVID-19.