The effects of cuproptosis on TIME revealed a higher expression of chemokines in Cluster 1 in TCGA, meta, LGG, and glioblastoma cohorts, such as CXCL12, CXCL10, CCR5, CCR10, CCL5, and CCR2 (Figure 2A), attracting immunosuppressive cells, such as Tregs, macrophages, myeloid‐derived suppressor cells (MDSCs), and monocytes, vital in immune escape. Here, CCR10 is linked to glioblastoma.