Analysis of the G5P[7] profile indicated that infection with this virus downregulated pathways associated with the expression of tyrosine kinases (FAK), the dominant negative inhibitor of basic helix-loop-helix proteins (ID1) [46], the growth of the intestinal mucosa (growth hormone) [47], mediators involved in the regulation of epithelial structure/function (eicosanoid) [48], molecules associated with cancer development (breast cancer regulation by stathmin 1) [49] and sperm motility. This evidence concerns the gene PTK2 and breast cancer.