PARP9 and coronary artery disorder: Proteome analyses from stimulated and non-stimulated human and mouse macrophage-like cells detected an increase in the ADP-ribosylated PARP14/ARTD8 and PARP9/ARTD9 peptide levels upon stimulation with IFN-γ, and network analysis identified a close link between those PARPs/ARTs and the human coronary artery disease gene module [79,80].