TP53 and acute lymphoblastic leukemia: Other less common mechanisms including Bcl-2 suppression through the p53/miR-34a axis on MM cells [59], wt-p53 accumulation on wt-p53 Molt-4 cells ALL cell line [60], inhibition of Akt/mTOR pathways through PPARγ/PTEN axis on MCF-7 cells [61], activation of acyl-CoA synthetase (ACS) on lymphoma cell line Ramos [62], and displacement of ARA from tumor-cell membranes after omega-3 PUFAs treatment on in vivo model [63].