ADGRL3 and viral infectious disease: These included PRX, TRX, HDM, CL1 and CL3, major components of the FhNEJ secretome that play an important role in the modulation of innate immune responses [30,31,32]; KTM, Cys1 and KTSPIDP, which are cathepsin protease inhibitors [33,34,35] that can be relevant during viral infection processes [36]; and VGCH1, CRTA and CAL, which are tegument proteins important for FhNEJ biology and potentially involved in host–parasite interactions [20].