NFKB1 and neoplasm: For example, Aravindan et al. showed that LDIR can induce an adaptive response via the activation of NF-κB dependent responsive tumor necrosis factor α (TNF-α), interleukin 1a, cMYC, and SOD2 via intercellular communication and sequential orchestration, endorsing radiation protection (radioresistance) of surviving tumor cells [408].