The overexpression of miR-205 inhibits glioma cells’ invasive behavior and improves apoptosis and cell-cycle arrest at the G1/G0 phase by targeting vascular endothelial growth factor A (VEGF A) (Figure 2F and Figure 4F) [119], which is the most potent mediator of angiogenesis in glioma [121]. Here, VEGFA is linked to glioma.