PRDX3 and Behcet disease: β-OHB plays a role in suppressing oxidative stress induced by oxidant treatment through increasing endogenous antioxidant capacities [5], and we report that BD enhances the whole tissue antioxidant soluble capacity, promotes the overexpression of proteins involved in enzymatic antioxidant capacity (SOD-2, PRDX-3, and CAT), and reduces tissue ROS content and the oxidative damages to lipids and proteins.