In this context, failures in GLAST, GLT-1, and EAAC1 transporters in epilepsy, in addition to collaborating with the excitotoxicity of the glutamatergic system, provoke failures in the production of neuronal GSH, justifying the reduction of GSH in epileptic animals observed in the meta-analysis [28,91]. The gene discussed is SLC1A2; the disease is epilepsy.