ACO1 and bronchopulmonary dysplasia: In addition, enrichment in pathways for IFN signaling and cytokine ‘storm’ (e.g., IFI6, IFITM1, IL1RN, MYD88, LCK), antioxidant response regulated by nuclear factor erythroid 2-related factor 2/glutathione redox (e.g., CHST12, GCLM, GSR, NFE2L2, SOD1), and mitochondrial dysfunction (e.g., ACO1, COX7B, NDUFS4) indicated severe oxidative stress and inflammation in infants who developed BPD.