As Koyama et al.23investigated the consequence of STK11/LKB1 deficiency on the TIME in a mouse model of KRAS‐driven NSCLC, and they confirmed that STK11/LKB1 deficiency promotes neutrophil recruitment and proinflammatory cytokine production to suppress T‐cell activity in the lung TME. This evidence concerns the gene STK11 and non-small cell lung carcinoma.