In order to elucidate the molecular mechanisms underlying the ameliorative effects of FGF10 on AD‐like symptoms and pathologies, we conducted an analysis of the phosphorylation and total levels of FGFR1, FGFR2, FGFR3, Fibroblast Growth Factor Receptor Substrate 2 (FRS2), and AKT. This evidence concerns the gene AKT1 and Alzheimer disease.