FGF10 and Alzheimer disease: Collectively, based on the findings from the in vivo and in vitro AD models, we confirmed that FGF10 has the potential to mitigate neuronal damage and synaptic deficits in 3xTg‐AD mice and APPswe‐transfected HT22 cells, suggesting the potential of FGF10 as a promising intervention for restoring synaptic dysfunction in the context of AD.