Some mechanisms for viral cells invasion have been hypothesized, such as the role of angiotensin-converting enzyme 2 (ACE2) receptors, scavengers receptor B type 1 (SR-B1) and other cellular wall receptors that allow the entry of the virus into the endothelial cells, thus determining endothelial dysfunction and endothelialitis. Here, SCARB1 is linked to endothelial dysfunction.