SLC7A11 and glioblastoma: For example, NF-κB-activating protein (NKAP) has been discovered to directly bind to m6A-methylated SLC7A11 mRNA and then recruit the splicing factor proline and glutamine-rich (SFPQ), which recognize the splice site and thus promotes SLC7A11 mRNA splicing and maturation, ultimately preventing the ferroptosis of glioblastoma cells163.